"When the body recognizes a viral infection, our immune system initiates the production of interferons. Interferons are a group of cytokines that help shape the immune response and are therefore essential in the fight against a viral infection. Alveolar macrophages have previously been shown to produce large amounts of interferons upon infection with respiratory viruses, such as influenza.
SARS-CoV-2 is a respiratory virus that typically infects the outermost cell layer of the lungs, the epithelial layer. We now know that the ACE receptors are the target of the spike protein and the epithelium of the arteries are also a vulnerable tissue for entry of the Coronavirus 2. New research has shown that interferon production in the infected epithelial cells can be inhibited by the SARS-CoV-2 virus. This results in low interferon production and therefore also a limited activation of the immune system to fight against the virus. Although the epithelial layer is the target of the virus, it must be assumed that the first cell type the virus encounters is the alveolar macrophages, and therefore these cells are important for how quickly an immune response to a SARS-CoV-2 infection can be initiated." (Louise Dalskov et al, SARS‐CoV‐2 evades immune detection in alveolar macrophages, EMBO reports (2020). DOI: 10.15252/embr.202051252)
Epithelial cells lining the nose and throat are the first line of defense against any airborne virus carried through the air by aerosols from an infected person. The lungs and it's alveoli are the very next landing site and thus, line of defense, next to the blood vessels themselves.
aerosols: infectious viral particles that can float or drift around in the air. Aerosols are emitted by a person infected with coronavirus — even one with no symptoms — when they talk, breathe, cough, or sneeze. Another person can breathe in these aerosols and become infected with the virus. (health.harvard.edu)
The first step of an Immune Response to a new viral invasion is: Increasing the activation and expression of genes involved with anti-viral responses.
How can one stimulate and speed up this process bioenergetically!
Interferons are the first responders to any viral contact. In the case of an acute contact this would be Interferon 1 and Interferon 2. Interferon Alpha (IFNAR1) is the receptor for Interferon 1, IFNAR2 is the receptor for interferon 2.
Interferons are part of the cytokines immune signaling proteins of the body. There are several groups of interferons, since they don't just deal with viruses, but other pathogens as well, and the complex inflammatory responses. All these different immune factors communicate with each other through their functional genes to achieve the best immune response. In the case of viruses, the Interferon 1 and 2 are the most critical to stimulate early on, since it directly competes with viruses (at the cell membrane receptor level), for access to transcription in the nucleus (RNA copying). SARS2 is particularly aggressive in blocking the Interferon 1 receptor in favor of it's own passage through the receptor toward the cell nucleus.
I feel that a biofeedback of the involved immune factors can be helpful in support of a speedy immune response.
JAK1, STAT1 and STAT2 are part of the intra-cellular response, controlling access to the cell nucleus through the cell membrane.
Interferon 1 and Interferon 2 or IFR 1 and IFR 2 are the most fundamental factors. Type them into the search function of the IMAET System Allergen Profile and they will show on the Comprehensive Database window.
Put the desired items into the custom treatment data 'basket', add all additional feedback items and set the timer at 20 minutes. Activating the Quick Cycle is a good idea.
Communicating with the Immune System is possible. The details necessary to make an effective communication possible is at your fingertips with the IMAET data base.
When we're performing a biofeedback with interferon and other immune factors, we are resonating with the genes that are expressing these factors. This could potentially upregulate their expression for better and quicker function. And that pushes the scales towards increasing interferon production and inhibiting virus replication, thus shortening the course and severity of the infection.